Electrophysiological Abnormalities of the Atrial Muscle in Patients with Manifest Wolff‐Parkinson‐White Syndrome Associated with Paroxysmal Atrial Fibrillation
Identifieur interne : 000013 ( Main/Corpus ); précédent : 000012; suivant : 000014Electrophysiological Abnormalities of the Atrial Muscle in Patients with Manifest Wolff‐Parkinson‐White Syndrome Associated with Paroxysmal Atrial Fibrillation
Auteurs : Atsushi Konoe ; Masahiko Fukatani ; Muneo Tanigawa ; Shohro Isomoto ; Mitsuo Kadena ; Toshifumi Sakamoto ; Mitsuhiro Mori ; Akihiko Shimizu ; Kunitake HashibaSource :
- Pacing and Clinical Electrophysiology [ 0147-8389 ] ; 1992-07.
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- KwdEn :
Abstract
We investigated the electrophysiological properties of the atrial muscle in 33 patients with manifest Wolff‐Parkinson‐White syndrome. Group I consisted of 13 patients with paroxysmal atrial fibrillation and group II consisted of 20 patients without paroxysmal atrial fibrillation. The anterograde and retrograde effective refractory periods of the accessory pathway and the inducibility of atrioventricular reciprocating tachycardia were not significantly different between the two groups, Endocardial electrograms, obtained by right atrial catheter mapping, were recorded during sinus rhythm from 12 sites of the right atrium in 12 of the 13 group I patients and in all group II patients. An abnormal atrial electrogram was defined as 100 msec or longer in duration, and/or the occurrence of eight or more deflections. Ten (83%) of the 12 group I patients had abnormal atrial electrograms, while only two (10%) of the 20 group II patients had abnormal atrial electrograms, and the difference was significant (P < 0.01). Thirty‐six (26%) of the total 139 electrograms obtained from 12 group I patients and two (1%) of the total 199 electrograms obtained from 20 group II patients fulfilled the criteria for an abnormal atrial electrogram, and the difference was significant (P < 0.01). The fragmented atrial activity zone, interatrial conduction delay zone, and repetitive atrial firing zone obtained by right atrial extrastimulation were significantly wider in group I than in group II, respectively. It was concluded that electrical abnormalities of the atrial muscle may play an important role in the occurrence of paroxysmal atrial fibrillation in patients with Wolff‐Parkinson‐White syndrome.
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DOI: 10.1111/j.1540-8159.1992.tb03098.x
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Nagasaki University School of Medicine, Nagasaki, Japan</mods:affiliation></affiliation></author><author><name sortKey="Mori, Mitsuhiro" sort="Mori, Mitsuhiro" uniqKey="Mori M" first="Mitsuhiro" last="Mori">Mitsuhiro Mori</name><affiliation><mods:affiliation>Third Department of Internal Medicine, Nagasaki University School of Medicine, Nagasaki, Japan</mods:affiliation></affiliation></author><author><name sortKey="Shimizu, Akihiko" sort="Shimizu, Akihiko" uniqKey="Shimizu A" first="Akihiko" last="Shimizu">Akihiko Shimizu</name><affiliation><mods:affiliation>Third Department of Internal Medicine, Nagasaki University School of Medicine, Nagasaki, Japan</mods:affiliation></affiliation></author><author><name sortKey="Hashiba, Kunitake" sort="Hashiba, Kunitake" uniqKey="Hashiba K" first="Kunitake" last="Hashiba">Kunitake Hashiba</name><affiliation><mods:affiliation>Third Department of Internal Medicine, Nagasaki University School of Medicine, Nagasaki, 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Japan</mods:affiliation></affiliation></author><author><name sortKey="Fukatani, Masahiko" sort="Fukatani, Masahiko" uniqKey="Fukatani M" first="Masahiko" last="Fukatani">Masahiko Fukatani</name><affiliation><mods:affiliation>Third Department of Internal Medicine, Nagasaki University School of Medicine, Nagasaki, Japan</mods:affiliation></affiliation></author><author><name sortKey="Tanigawa, Muneo" sort="Tanigawa, Muneo" uniqKey="Tanigawa M" first="Muneo" last="Tanigawa">Muneo Tanigawa</name><affiliation><mods:affiliation>Third Department of Internal Medicine, Nagasaki University School of Medicine, Nagasaki, Japan</mods:affiliation></affiliation></author><author><name sortKey="Isomoto, Shohro" sort="Isomoto, Shohro" uniqKey="Isomoto S" first="Shohro" last="Isomoto">Shohro Isomoto</name><affiliation><mods:affiliation>Third Department of Internal Medicine, Nagasaki University School of Medicine, Nagasaki, Japan</mods:affiliation></affiliation></author><author><name sortKey="Kadena, Mitsuo" sort="Kadena, Mitsuo" uniqKey="Kadena M" first="Mitsuo" last="Kadena">Mitsuo Kadena</name><affiliation><mods:affiliation>Third Department of Internal Medicine, Nagasaki University School of Medicine, Nagasaki, Japan</mods:affiliation></affiliation></author><author><name sortKey="Sakamoto, Toshifumi" sort="Sakamoto, Toshifumi" uniqKey="Sakamoto T" first="Toshifumi" last="Sakamoto">Toshifumi Sakamoto</name><affiliation><mods:affiliation>Third Department of Internal Medicine, Nagasaki University School of Medicine, Nagasaki, Japan</mods:affiliation></affiliation></author><author><name sortKey="Mori, Mitsuhiro" sort="Mori, Mitsuhiro" uniqKey="Mori M" first="Mitsuhiro" last="Mori">Mitsuhiro Mori</name><affiliation><mods:affiliation>Third Department of Internal Medicine, Nagasaki University School of Medicine, Nagasaki, Japan</mods:affiliation></affiliation></author><author><name sortKey="Shimizu, Akihiko" sort="Shimizu, Akihiko" uniqKey="Shimizu A" first="Akihiko" last="Shimizu">Akihiko Shimizu</name><affiliation><mods:affiliation>Third Department of Internal Medicine, Nagasaki University School of Medicine, Nagasaki, Japan</mods:affiliation></affiliation></author><author><name sortKey="Hashiba, Kunitake" sort="Hashiba, Kunitake" uniqKey="Hashiba K" first="Kunitake" last="Hashiba">Kunitake Hashiba</name><affiliation><mods:affiliation>Third Department of Internal Medicine, Nagasaki University School of Medicine, Nagasaki, Japan</mods:affiliation></affiliation></author></analytic><monogr></monogr><series><title level="j">Pacing and Clinical Electrophysiology</title><idno type="ISSN">0147-8389</idno><idno type="eISSN">1540-8159</idno><imprint><publisher>Blackwell Publishing Ltd</publisher><pubPlace>Oxford, UK</pubPlace><date type="published" when="1992-07">1992-07</date><biblScope unit="volume">15</biblScope><biblScope unit="issue">7</biblScope><biblScope unit="page" from="1040">1040</biblScope><biblScope unit="page" 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Group I consisted of 13 patients with paroxysmal atrial fibrillation and group II consisted of 20 patients without paroxysmal atrial fibrillation. The anterograde and retrograde effective refractory periods of the accessory pathway and the inducibility of atrioventricular reciprocating tachycardia were not significantly different between the two groups, Endocardial electrograms, obtained by right atrial catheter mapping, were recorded during sinus rhythm from 12 sites of the right atrium in 12 of the 13 group I patients and in all group II patients. An abnormal atrial electrogram was defined as 100 msec or longer in duration, and/or the occurrence of eight or more deflections. Ten (83%) of the 12 group I patients had abnormal atrial electrograms, while only two (10%) of the 20 group II patients had abnormal atrial electrograms, and the difference was significant (P < 0.01). Thirty‐six (26%) of the total 139 electrograms obtained from 12 group I patients and two (1%) of the total 199 electrograms obtained from 20 group II patients fulfilled the criteria for an abnormal atrial electrogram, and the difference was significant (P < 0.01). The fragmented atrial activity zone, interatrial conduction delay zone, and repetitive atrial firing zone obtained by right atrial extrastimulation were significantly wider in group I than in group II, respectively. It was concluded that electrical abnormalities of the atrial muscle may play an important role in the occurrence of paroxysmal atrial fibrillation in patients with Wolff‐Parkinson‐White syndrome.</div></front></TEI><istex><corpusName>wiley</corpusName><author><json:item><name>ATSUSHI KONOE</name><affiliations><json:string>Third Department of Internal Medicine, Nagasaki University School of Medicine, Nagasaki, Japan</json:string></affiliations></json:item><json:item><name>MASAHIKO FUKATANI</name><affiliations><json:string>Third Department of Internal Medicine, Nagasaki University School of Medicine, Nagasaki, Japan</json:string></affiliations></json:item><json:item><name>MUNEO TANIGAWA</name><affiliations><json:string>Third Department of Internal Medicine, Nagasaki University School of Medicine, Nagasaki, Japan</json:string></affiliations></json:item><json:item><name>SHOHRO ISOMOTO</name><affiliations><json:string>Third Department of Internal Medicine, Nagasaki University School of Medicine, Nagasaki, Japan</json:string></affiliations></json:item><json:item><name>MITSUO KADENA</name><affiliations><json:string>Third Department of Internal Medicine, Nagasaki University School of Medicine, Nagasaki, Japan</json:string></affiliations></json:item><json:item><name>TOSHIFUMI SAKAMOTO</name><affiliations><json:string>Third Department of Internal Medicine, Nagasaki University School of Medicine, Nagasaki, Japan</json:string></affiliations></json:item><json:item><name>MITSUHIRO MORI</name><affiliations><json:string>Third Department of Internal Medicine, Nagasaki University School of Medicine, Nagasaki, Japan</json:string></affiliations></json:item><json:item><name>AKIHIKO SHIMIZU</name><affiliations><json:string>Third Department of Internal Medicine, Nagasaki University School of Medicine, Nagasaki, Japan</json:string></affiliations></json:item><json:item><name>KUNITAKE HASHIBA</name><affiliations><json:string>Third Department of Internal Medicine, Nagasaki University School of Medicine, Nagasaki, Japan</json:string></affiliations></json:item></author><subject><json:item><lang><json:string>eng</json:string></lang><value>paroxysmal atrial fibrillation</value></json:item><json:item><lang><json:string>eng</json:string></lang><value>manifest Wolff‐Parkinson‐White syndrome</value></json:item><json:item><lang><json:string>eng</json:string></lang><value>atrial vulnerability</value></json:item><json:item><lang><json:string>eng</json:string></lang><value>right atrial mapping during sinus rhythm</value></json:item><json:item><lang><json:string>eng</json:string></lang><value>prolonged and fractionated right atrial electrogram</value></json:item></subject><articleId><json:string>PACE1040</json:string></articleId><language><json:string>eng</json:string></language><abstract>We investigated the electrophysiological properties of the atrial muscle in 33 patients with manifest Wolff‐Parkinson‐White syndrome. Group I consisted of 13 patients with paroxysmal atrial fibrillation and group II consisted of 20 patients without paroxysmal atrial fibrillation. The anterograde and retrograde effective refractory periods of the accessory pathway and the inducibility of atrioventricular reciprocating tachycardia were not significantly different between the two groups, Endocardial electrograms, obtained by right atrial catheter mapping, were recorded during sinus rhythm from 12 sites of the right atrium in 12 of the 13 group I patients and in all group II patients. An abnormal atrial electrogram was defined as 100 msec or longer in duration, and/or the occurrence of eight or more deflections. Ten (83%) of the 12 group I patients had abnormal atrial electrograms, while only two (10%) of the 20 group II patients had abnormal atrial electrograms, and the difference was significant (P > 0.01). Thirty‐six (26%) of the total 139 electrograms obtained from 12 group I patients and two (1%) of the total 199 electrograms obtained from 20 group II patients fulfilled the criteria for an abnormal atrial electrogram, and the difference was significant (P > 0.01). The fragmented atrial activity zone, interatrial conduction delay zone, and repetitive atrial firing zone obtained by right atrial extrastimulation were significantly wider in group I than in group II, respectively. It was concluded that electrical abnormalities of the atrial muscle may play an important role in the occurrence of paroxysmal atrial fibrillation in patients with Wolff‐Parkinson‐White syndrome.</abstract><qualityIndicators><score>9.48</score><pdfVersion>1.6</pdfVersion><pdfPageSize>567 x 794 pts</pdfPageSize><refBibsNative>true</refBibsNative><keywordCount>5</keywordCount><abstractCharCount>1697</abstractCharCount><pdfWordCount>4980</pdfWordCount><pdfCharCount>32862</pdfCharCount><pdfPageCount>14</pdfPageCount><abstractWordCount>252</abstractWordCount></qualityIndicators><title>Electrophysiological Abnormalities of the Atrial Muscle in Patients with Manifest Wolff‐Parkinson‐White Syndrome Associated with Paroxysmal Atrial Fibrillation</title><genre><json:string>article</json:string></genre><host><volume>15</volume><publisherId><json:string>PACE</json:string></publisherId><pages><total>13</total><last>1052</last><first>1040</first></pages><issn><json:string>0147-8389</json:string></issn><issue>7</issue><genre><json:string>Journal</json:string></genre><language><json:string>unknown</json:string></language><eissn><json:string>1540-8159</json:string></eissn><title>Pacing and Clinical Electrophysiology</title><doi><json:string>10.1111/(ISSN)1540-8159</json:string></doi></host><publicationDate>1992</publicationDate><copyrightDate>1992</copyrightDate><doi><json:string>10.1111/j.1540-8159.1992.tb03098.x</json:string></doi><id>7017D4570014BC872FE82A5636E0A706E8A67BB1</id><fulltext><json:item><original>true</original><mimetype>application/pdf</mimetype><extension>pdf</extension><uri>https://api.istex.fr/document/7017D4570014BC872FE82A5636E0A706E8A67BB1/fulltext/pdf</uri></json:item><json:item><original>false</original><mimetype>application/zip</mimetype><extension>zip</extension><uri>https://api.istex.fr/document/7017D4570014BC872FE82A5636E0A706E8A67BB1/fulltext/zip</uri></json:item><istex:fulltextTEI uri="https://api.istex.fr/document/7017D4570014BC872FE82A5636E0A706E8A67BB1/fulltext/tei"><teiHeader><fileDesc><titleStmt><title level="a" type="main" xml:lang="en">Electrophysiological Abnormalities of the Atrial Muscle in Patients with Manifest Wolff‐Parkinson‐White Syndrome Associated with Paroxysmal Atrial Fibrillation</title></titleStmt><publicationStmt><authority>ISTEX</authority><publisher>Blackwell Publishing Ltd</publisher><pubPlace>Oxford, UK</pubPlace><availability><p>WILEY</p></availability><date>1992</date></publicationStmt><sourceDesc><biblStruct type="inbook"><analytic><title level="a" type="main" xml:lang="en">Electrophysiological Abnormalities of the Atrial Muscle in Patients with Manifest Wolff‐Parkinson‐White Syndrome Associated with Paroxysmal Atrial Fibrillation</title><author><persName><forename type="first">ATSUSHI</forename><surname>KONOE</surname></persName><note type="correspondence"><p>Correspondence: Address for reprints: Atsushi Konoe, M.D., The Third Department of Internal Medicine, Nagasaki University School of Medicine. 7–1 Sakamoto‐machi, Nagasaki 852, Japan. Fax: 0958–46–2500.</p></note><affiliation>Third Department of Internal Medicine, Nagasaki University School of Medicine, Nagasaki, Japan</affiliation></author><author><persName><forename type="first">MASAHIKO</forename><surname>FUKATANI</surname></persName><affiliation>Third Department of Internal Medicine, Nagasaki University School of Medicine, Nagasaki, Japan</affiliation></author><author><persName><forename type="first">MUNEO</forename><surname>TANIGAWA</surname></persName><affiliation>Third Department of Internal Medicine, Nagasaki University School of Medicine, Nagasaki, Japan</affiliation></author><author><persName><forename type="first">SHOHRO</forename><surname>ISOMOTO</surname></persName><affiliation>Third Department of Internal Medicine, Nagasaki University School of Medicine, Nagasaki, Japan</affiliation></author><author><persName><forename type="first">MITSUO</forename><surname>KADENA</surname></persName><affiliation>Third Department of Internal Medicine, Nagasaki University School of Medicine, Nagasaki, Japan</affiliation></author><author><persName><forename type="first">TOSHIFUMI</forename><surname>SAKAMOTO</surname></persName><affiliation>Third Department of Internal Medicine, Nagasaki University School of Medicine, Nagasaki, Japan</affiliation></author><author><persName><forename type="first">MITSUHIRO</forename><surname>MORI</surname></persName><affiliation>Third Department of Internal Medicine, Nagasaki University School of Medicine, Nagasaki, Japan</affiliation></author><author><persName><forename type="first">AKIHIKO</forename><surname>SHIMIZU</surname></persName><affiliation>Third Department of Internal Medicine, Nagasaki University School of Medicine, Nagasaki, Japan</affiliation></author><author><persName><forename type="first">KUNITAKE</forename><surname>HASHIBA</surname></persName><affiliation>Third Department of Internal Medicine, Nagasaki University School of Medicine, Nagasaki, Japan</affiliation></author></analytic><monogr><title level="j">Pacing and Clinical Electrophysiology</title><idno type="pISSN">0147-8389</idno><idno type="eISSN">1540-8159</idno><idno type="DOI">10.1111/(ISSN)1540-8159</idno><imprint><publisher>Blackwell Publishing Ltd</publisher><pubPlace>Oxford, UK</pubPlace><date type="published" when="1992-07"></date><biblScope unit="volume">15</biblScope><biblScope unit="issue">7</biblScope><biblScope unit="page" from="1040">1040</biblScope><biblScope unit="page" to="1052">1052</biblScope></imprint></monogr><idno type="istex">7017D4570014BC872FE82A5636E0A706E8A67BB1</idno><idno type="DOI">10.1111/j.1540-8159.1992.tb03098.x</idno><idno type="ArticleID">PACE1040</idno></biblStruct></sourceDesc></fileDesc><profileDesc><creation><date>1992</date></creation><langUsage><language ident="en">en</language></langUsage><abstract xml:lang="en"><p>We investigated the electrophysiological properties of the atrial muscle in 33 patients with manifest Wolff‐Parkinson‐White syndrome. Group I consisted of 13 patients with paroxysmal atrial fibrillation and group II consisted of 20 patients without paroxysmal atrial fibrillation. The anterograde and retrograde effective refractory periods of the accessory pathway and the inducibility of atrioventricular reciprocating tachycardia were not significantly different between the two groups, Endocardial electrograms, obtained by right atrial catheter mapping, were recorded during sinus rhythm from 12 sites of the right atrium in 12 of the 13 group I patients and in all group II patients. An abnormal atrial electrogram was defined as 100 msec or longer in duration, and/or the occurrence of eight or more deflections. Ten (83%) of the 12 group I patients had abnormal atrial electrograms, while only two (10%) of the 20 group II patients had abnormal atrial electrograms, and the difference was significant (P < 0.01). Thirty‐six (26%) of the total 139 electrograms obtained from 12 group I patients and two (1%) of the total 199 electrograms obtained from 20 group II patients fulfilled the criteria for an abnormal atrial electrogram, and the difference was significant (P < 0.01). The fragmented atrial activity zone, interatrial conduction delay zone, and repetitive atrial firing zone obtained by right atrial extrastimulation were significantly wider in group I than in group II, respectively. It was concluded that electrical abnormalities of the atrial muscle may play an important role in the occurrence of paroxysmal atrial fibrillation in patients with Wolff‐Parkinson‐White syndrome.</p></abstract><textClass xml:lang="en"><keywords scheme="keyword"><list><head>Keywords</head><item><term>paroxysmal atrial fibrillation</term></item><item><term>manifest Wolff‐Parkinson‐White syndrome</term></item><item><term>atrial vulnerability</term></item><item><term>right atrial mapping during sinus rhythm</term></item><item><term>prolonged and fractionated right atrial electrogram</term></item></list></keywords></textClass></profileDesc><revisionDesc><change when="1992-07">Published</change></revisionDesc></teiHeader></istex:fulltextTEI><json:item><original>false</original><mimetype>text/plain</mimetype><extension>txt</extension><uri>https://api.istex.fr/document/7017D4570014BC872FE82A5636E0A706E8A67BB1/fulltext/txt</uri></json:item></fulltext><metadata><istex:metadataXml wicri:clean="Wiley, elements deleted: body"><istex:xmlDeclaration>version="1.0" encoding="UTF-8" standalone="yes"</istex:xmlDeclaration><istex:document><component version="2.0" type="serialArticle" xml:lang="en"><header><publicationMeta level="product"><publisherInfo><publisherName>Blackwell Publishing Ltd</publisherName><publisherLoc>Oxford, UK</publisherLoc></publisherInfo><doi origin="wiley" registered="yes">10.1111/(ISSN)1540-8159</doi><issn type="print">0147-8389</issn><issn type="electronic">1540-8159</issn><idGroup><id type="product" value="PACE"></id><id type="publisherDivision" value="ST"></id></idGroup><titleGroup><title type="main" sort="PACING CLINICAL ELECTROPHYSIOLOGY">Pacing and Clinical Electrophysiology</title></titleGroup></publicationMeta><publicationMeta level="part" position="07007"><doi origin="wiley">10.1111/pace.1992.15.issue-7</doi><numberingGroup><numbering type="journalVolume" number="15">15</numbering><numbering type="journalIssue" number="7">7</numbering></numberingGroup><coverDate startDate="1992-07">July 1992</coverDate></publicationMeta><publicationMeta level="unit" type="article" position="0104000" status="forIssue"><doi origin="wiley">10.1111/j.1540-8159.1992.tb03098.x</doi><idGroup><id type="unit" value="PACE1040"></id></idGroup><countGroup><count type="pageTotal" number="13"></count></countGroup><titleGroup><title type="tocHeading1">ORIGINAL ARTICLES</title></titleGroup><eventGroup><event type="firstOnline" date="2006-06-30"></event><event type="publishedOnlineFinalForm" date="2006-06-30"></event><event type="xmlConverted" agent="Converter:BPG_TO_WML3G version:2.3.2 mode:FullText source:HeaderRef result:HeaderRef" date="2010-03-02"></event><event type="xmlConverted" agent="Converter:WILEY_ML3G_TO_WILEY_ML3GV2 version:3.8.8" date="2014-02-06"></event><event type="xmlConverted" agent="Converter:WML3G_To_WML3G version:4.1.7 mode:FullText,remove_FC" date="2014-11-03"></event></eventGroup><numberingGroup><numbering type="pageFirst" number="1040">1040</numbering><numbering type="pageLast" number="1052">1052</numbering></numberingGroup><correspondenceTo>Address for reprints: Atsushi Konoe, M.D., The Third Department of Internal Medicine, Nagasaki University School of Medicine. 7–1 Sakamoto‐machi, Nagasaki 852, Japan. Fax: 0958–46–2500.</correspondenceTo><linkGroup><link type="toTypesetVersion" href="file:PACE.PACE1040.pdf"></link></linkGroup></publicationMeta><contentMeta><unparsedEditorialHistory>Received November 7, 1990; revision September 9, 1991; revision January 10, 1992; accepted January 30, 1992.</unparsedEditorialHistory><countGroup><count type="referenceTotal" number="28"></count><count type="linksCrossRef" number="10"></count></countGroup><titleGroup><title type="main">Electrophysiological Abnormalities of the Atrial Muscle in Patients with Manifest Wolff‐Parkinson‐White Syndrome Associated with Paroxysmal Atrial Fibrillation</title></titleGroup><creators><creator creatorRole="author" xml:id="cr1" affiliationRef="#a1" corresponding="yes"><personName><givenNames>ATSUSHI</givenNames><familyName>KONOE</familyName></personName></creator><creator creatorRole="author" xml:id="cr2" affiliationRef="#a1"><personName><givenNames>MASAHIKO</givenNames><familyName>FUKATANI</familyName></personName></creator><creator creatorRole="author" xml:id="cr3" affiliationRef="#a1"><personName><givenNames>MUNEO</givenNames><familyName>TANIGAWA</familyName></personName></creator><creator creatorRole="author" xml:id="cr4" affiliationRef="#a1"><personName><givenNames>SHOHRO</givenNames><familyName>ISOMOTO</familyName></personName></creator><creator creatorRole="author" xml:id="cr5" affiliationRef="#a1"><personName><givenNames>MITSUO</givenNames><familyName>KADENA</familyName></personName></creator><creator creatorRole="author" xml:id="cr6" affiliationRef="#a1"><personName><givenNames>TOSHIFUMI</givenNames><familyName>SAKAMOTO</familyName></personName></creator><creator creatorRole="author" xml:id="cr7" affiliationRef="#a1"><personName><givenNames>MITSUHIRO</givenNames><familyName>MORI</familyName></personName></creator><creator creatorRole="author" xml:id="cr8" affiliationRef="#a1"><personName><givenNames>AKIHIKO</givenNames><familyName>SHIMIZU</familyName></personName></creator><creator creatorRole="author" xml:id="cr9" affiliationRef="#a1"><personName><givenNames>KUNITAKE</givenNames><familyName>HASHIBA</familyName></personName></creator></creators><affiliationGroup><affiliation xml:id="a1" countryCode="JP"><unparsedAffiliation>Third Department of Internal Medicine, Nagasaki University School of Medicine, Nagasaki, Japan</unparsedAffiliation></affiliation></affiliationGroup><keywordGroup xml:lang="en"><keyword xml:id="k1">paroxysmal atrial fibrillation</keyword><keyword xml:id="k2">manifest Wolff‐Parkinson‐White syndrome</keyword><keyword xml:id="k3">atrial vulnerability</keyword><keyword xml:id="k4">right atrial mapping during sinus rhythm</keyword><keyword xml:id="k5">prolonged and fractionated right atrial electrogram</keyword></keywordGroup><abstractGroup><abstract type="main" xml:lang="en"><p>We investigated the electrophysiological properties of the atrial muscle in 33 patients with manifest Wolff‐Parkinson‐White syndrome. Group I consisted of 13 patients with paroxysmal atrial fibrillation and group II consisted of 20 patients without paroxysmal atrial fibrillation. The anterograde and retrograde effective refractory periods of the accessory pathway and the inducibility of atrioventricular reciprocating tachycardia were not significantly different between the two groups, Endocardial electrograms, obtained by right atrial catheter mapping, were recorded during sinus rhythm from 12 sites of the right atrium in 12 of the 13 group I patients and in all group II patients. An abnormal atrial electrogram was defined as 100 msec or longer in duration, and/or the occurrence of eight or more deflections. Ten (83%) of the 12 group I patients had abnormal atrial electrograms, while only two (10%) of the 20 group II patients had abnormal atrial electrograms, and the difference was significant (P < 0.01). Thirty‐six (26%) of the total 139 electrograms obtained from 12 group I patients and two (1%) of the total 199 electrograms obtained from 20 group II patients fulfilled the criteria for an abnormal atrial electrogram, and the difference was significant (P < 0.01). The fragmented atrial activity zone, interatrial conduction delay zone, and repetitive atrial firing zone obtained by right atrial extrastimulation were significantly wider in group I than in group II, respectively. It was concluded that electrical abnormalities of the atrial muscle may play an important role in the occurrence of paroxysmal atrial fibrillation in patients with Wolff‐Parkinson‐White syndrome.</p></abstract></abstractGroup></contentMeta></header></component></istex:document></istex:metadataXml><mods version="3.6"><titleInfo lang="en"><title>Electrophysiological Abnormalities of the Atrial Muscle in Patients with Manifest Wolff‐Parkinson‐White Syndrome Associated with Paroxysmal Atrial Fibrillation</title></titleInfo><titleInfo type="alternative" contentType="CDATA" lang="en"><title>Electrophysiological Abnormalities of the Atrial Muscle in Patients with Manifest Wolff‐Parkinson‐White Syndrome Associated with Paroxysmal Atrial Fibrillation</title></titleInfo><name type="personal"><namePart type="given">ATSUSHI</namePart><namePart type="family">KONOE</namePart><affiliation>Third Department of Internal Medicine, Nagasaki University School of Medicine, Nagasaki, Japan</affiliation><description>Correspondence: Address for reprints: Atsushi Konoe, M.D., The Third Department of Internal Medicine, Nagasaki University School of Medicine. 7–1 Sakamoto‐machi, Nagasaki 852, Japan. Fax: 0958–46–2500.</description><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">MASAHIKO</namePart><namePart type="family">FUKATANI</namePart><affiliation>Third Department of Internal Medicine, Nagasaki University School of Medicine, Nagasaki, Japan</affiliation><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">MUNEO</namePart><namePart type="family">TANIGAWA</namePart><affiliation>Third Department of Internal Medicine, Nagasaki University School of Medicine, Nagasaki, Japan</affiliation><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">SHOHRO</namePart><namePart type="family">ISOMOTO</namePart><affiliation>Third Department of Internal Medicine, Nagasaki University School of Medicine, Nagasaki, Japan</affiliation><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">MITSUO</namePart><namePart type="family">KADENA</namePart><affiliation>Third Department of Internal Medicine, Nagasaki University School of Medicine, Nagasaki, Japan</affiliation><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">TOSHIFUMI</namePart><namePart type="family">SAKAMOTO</namePart><affiliation>Third Department of Internal Medicine, Nagasaki University School of Medicine, Nagasaki, Japan</affiliation><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">MITSUHIRO</namePart><namePart type="family">MORI</namePart><affiliation>Third Department of Internal Medicine, Nagasaki University School of Medicine, Nagasaki, Japan</affiliation><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">AKIHIKO</namePart><namePart type="family">SHIMIZU</namePart><affiliation>Third Department of Internal Medicine, Nagasaki University School of Medicine, Nagasaki, Japan</affiliation><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">KUNITAKE</namePart><namePart type="family">HASHIBA</namePart><affiliation>Third Department of Internal Medicine, Nagasaki University School of Medicine, Nagasaki, Japan</affiliation><role><roleTerm type="text">author</roleTerm></role></name><typeOfResource>text</typeOfResource><genre type="article" displayLabel="article"></genre><originInfo><publisher>Blackwell Publishing Ltd</publisher><place><placeTerm type="text">Oxford, UK</placeTerm></place><dateIssued encoding="w3cdtf">1992-07</dateIssued><edition>Received November 7, 1990; revision September 9, 1991; revision January 10, 1992; accepted January 30, 1992.</edition><copyrightDate encoding="w3cdtf">1992</copyrightDate></originInfo><language><languageTerm type="code" authority="rfc3066">en</languageTerm><languageTerm type="code" authority="iso639-2b">eng</languageTerm></language><physicalDescription><internetMediaType>text/html</internetMediaType><extent unit="references">28</extent></physicalDescription><abstract lang="en">We investigated the electrophysiological properties of the atrial muscle in 33 patients with manifest Wolff‐Parkinson‐White syndrome. Group I consisted of 13 patients with paroxysmal atrial fibrillation and group II consisted of 20 patients without paroxysmal atrial fibrillation. The anterograde and retrograde effective refractory periods of the accessory pathway and the inducibility of atrioventricular reciprocating tachycardia were not significantly different between the two groups, Endocardial electrograms, obtained by right atrial catheter mapping, were recorded during sinus rhythm from 12 sites of the right atrium in 12 of the 13 group I patients and in all group II patients. An abnormal atrial electrogram was defined as 100 msec or longer in duration, and/or the occurrence of eight or more deflections. Ten (83%) of the 12 group I patients had abnormal atrial electrograms, while only two (10%) of the 20 group II patients had abnormal atrial electrograms, and the difference was significant (P < 0.01). Thirty‐six (26%) of the total 139 electrograms obtained from 12 group I patients and two (1%) of the total 199 electrograms obtained from 20 group II patients fulfilled the criteria for an abnormal atrial electrogram, and the difference was significant (P < 0.01). The fragmented atrial activity zone, interatrial conduction delay zone, and repetitive atrial firing zone obtained by right atrial extrastimulation were significantly wider in group I than in group II, respectively. It was concluded that electrical abnormalities of the atrial muscle may play an important role in the occurrence of paroxysmal atrial fibrillation in patients with Wolff‐Parkinson‐White syndrome.</abstract><subject lang="en"><genre>Keywords</genre><topic>paroxysmal atrial fibrillation</topic><topic>manifest Wolff‐Parkinson‐White syndrome</topic><topic>atrial vulnerability</topic><topic>right atrial mapping during sinus rhythm</topic><topic>prolonged and fractionated right atrial electrogram</topic></subject><relatedItem type="host"><titleInfo><title>Pacing and Clinical Electrophysiology</title></titleInfo><genre type="Journal">journal</genre><identifier type="ISSN">0147-8389</identifier><identifier type="eISSN">1540-8159</identifier><identifier type="DOI">10.1111/(ISSN)1540-8159</identifier><identifier type="PublisherID">PACE</identifier><part><date>1992</date><detail type="volume"><caption>vol.</caption><number>15</number></detail><detail type="issue"><caption>no.</caption><number>7</number></detail><extent unit="pages"><start>1040</start><end>1052</end><total>13</total></extent></part></relatedItem><identifier type="istex">7017D4570014BC872FE82A5636E0A706E8A67BB1</identifier><identifier type="DOI">10.1111/j.1540-8159.1992.tb03098.x</identifier><identifier type="ArticleID">PACE1040</identifier><recordInfo><recordContentSource>WILEY</recordContentSource><recordOrigin>Blackwell Publishing Ltd</recordOrigin></recordInfo></mods></metadata><serie></serie></istex></record>Pour manipuler ce document sous Unix (Dilib)
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